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found that circ_0000808 was upregulated in NSCLC cells, where it enhanced the expression of the Gln transporter SLC1A5 by interacting with miR1827, thereby promoting Gln metabolism and facilitating the progression of NSCLC ( ncRNAs also influence tumor Gln metabolism by modulating various signaling pathways ( Table 1 )

Insulin-regulated hepatic gluconeogenesis through FOXO1-PGC-1alpha interaction

This atypical presentation of diabetic ketoacidosis is characterized by euglycemia (blood glucose less than 250 mg/dL), severe metabolic acidosis (pH less than 7.3, bicarbonate less than 18 mEq/L), and ketosis with ketonemia

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